PALI-2108: Completed Phase 1a Clinical Trial in Ulcerative Colitis & Fibrostenotic Crohn’s Disease
Double-Blind, Placebo-Controlled, Safety, Tolerability, PK and PD Study of PALI-2108 in NHV
Single Ascending Dose (n~40)
- Primary Endpoint: Safety and tolerability
- Secondary Endpoints: PK including Tmax, Cmax and T1/2
Multiple Ascending Dose (n~32)
- Primary Endpoint: Safety and tolerability
- Secondary Endpoints: PK including plasma and tissue C SS and colon: plasma ratio
Multiple Dose UC Cohort (n~5)
- Pharmacokinetic Endpoints: Plasma, colon tissue, urine, stool including plasma and tissue C SS and colon: plasma ratio
- Pharmacodynamic Endpoints: Fecal calpro, calpro epitope, hsCRP, colon tissue PDE4s, cAMP, TLC and histology
Topline Data Summary
GI-Restricted PDE4 Inhibitor Targeting Inflammation and Fibrosis in UC and Crohn’s Exceptional Safety & Tolerability
- 89 patients treated with no SAEs, no lab or EKG abnormalities and no TEAE-related discontinuations
- Completed all SAD cohorts: Single dose safe up to 450 mg
- Completed all MAD cohorts: BID dosing well tolerated up to 50 mg
- 15 mg BID had zero TEAEs, not even minor, in any subject
- Titration at 30 mg BID improved tolerability, with single minor TEAE, leveraging tachyphylaxis profile of PDE4s
Robust PK/PD Profile Supporting Mucosal Targeting
- Localized GI delivery confirmed: SI release and colonic bioactivation demonstrated in humans and preclinical models
- High mucosal drug levels exceeded IC₉₀ in colon, including 36h post-last dose
- Systemic exposure Cmax delayed and exposure right shifted and long half-life
- Long half-life and extended-release PK characteristics support QD dosing
Phase 1a Confirms PALI-2108 Was Safe and Well Tolerated
SAD Safety and Tolerability
MAD Safety and Tolerability
PALI-2018 Ph1a Findings:
- 84 patients treated and no SAEs and no AEs related to labs or EKGs
- Completed all SAD cohorts demonstrating Safety and Tolerability of single dose PALI-2108 up to 450mg
- Completed all MAD cohorts demonstrating safety and tolerability of twice daily (BID) doses of 15mg, 30mg and 50mg with 15mg BID having zero (not even minor) TEAE in any subject
- Completed FE cohort demonstrating minimal changes, reduced Cmax, right shifted AUC and delayed Tmax due to colon bioactivation
- Confirmed utility of titration with 30mg BID like other PDE4s
Phase 1 SAD/MAD PK Demonstrates PK Characteristics and Superior Exposure to Other PDE4s
Dose Proportionality and Extended Half-Life Enabling Daily Dosing
Single Ascending Dose (SAD)
PALI-2108 (prodrug; NOT SHOWN)
- Tmax: ~3–4h → consistent with small intestinal dissolution (Eudragit + prodrug design)
- Exposure (AUC): Minimal, as intended (GI-restricted)
PALI-0008 (active PDE4 inhibitor)
- Tmax: ~8–9h → consistent with ileocolonic bioconversion
- PK profile: Extended release; ~2–3× longer half-life than other PDE4 inhibitors (usually BID)
- Dose proportionality: Cmax and AUC approximately dose-proportional
Multiple Ascending Dose (MAD)
Bioconversion:
- PALI-2108 converted locally to PALI-0008 in terminal ileum/colon
Steady State:
- Reached within ~48h
- Stable pre-dose troughs with repeat dosing
- Pre-dose troughs ~20% higher than single-dose Cmax, above IC90
Dosing Implications:
- Extended half-life supports once-daily dosing
Phase 1 MAD Colon Tissue Pharmacokinetics
Tissue PK 36 Hours Post Dose Demonstrates Extended-Release PK and Potential for Daily Dosing
Tissue PK Observations from MAD
- Colon Tissue Concentrations approached IC90 36h post dose – demonstrating extended-release tissue PK at multiple dose levels supporting a daily dosing regimen
PALI-2108 Overview
Ulcerative Colitis: Completed PALI-2108 Phase 1b Clinical Trial
Fibrostenotic Crohn’s Disease: Completed PALI-2108 Phase 1b Clinical Trial
